返回
A synchronized design technique for efficient data distribution
DOI:10.1016/j.chb.2013.04.025.png)
摘要
En 中文
One of the important features of database fragmentation and allocation techniques is the fact that they depend not only on the entries of a database relation, but also on their empirical frequencies of use. Distributed processing is an effective way to improve performance of database systems. However, for a Distributed Database System (DDBS) to function efficiently, fragments of the database need to be allocated carefully at various sites across the relevant communications network. Therefore, fragmentation and proper allocation of fragments across network sites is considered as a key research area in distributed database environment. However, fragments allocation to the most appropriate sites is not an easy task to perform. This paper proposes a synchronized horizontal fragmentation, replication and allocation model that adopts a new approach to horizontally fragment a database relation based on attribute retrieval and update frequency to find an optimal solution for the allocation problem. A heuristic technique to satisfy horizontal fragmentation and allocation using a cost model to minimize the total cost of distribution is developed. Experimental results are consistent with the hypothesis and confirm that the proposed model can efficiently solve dynamic fragmentation and allocation problem in a distributed relational database environment. (C) 2013 Elsevier Ltd. All rights reserved.
Keyword:
Synchronization
Distribution
Fragmentation
Replication
Allocation
AI总结
对已上传原文的论文进行重点信息的提取,主要内容包括:简要概述、研究摘要、背景介绍、关键亮点、图文解析、展望与总结。
期刊
IF:
8.9
论文数:
9.0K
被引数:
5.8W
机构
暂无机构信息
引用论文
Placental glucocorticoid receptor and 11β-hydroxysteroid dehydrogenase-2 recruitment indicates impact of prenatal adversity upon postnatal development in mice
Stress
IF0
Overly Strong Priors for Socially Meaningful Visual Signals Are Linked to Psychosis Proneness in Healthy Individuals具有社会意义的视觉信号的过强先验与健康个体的精神病倾向有关
C/EBPα Short-Activating RNA Suppresses Metastasis of Hepatocellular Carcinoma through Inhibiting EGFR/β-Catenin Signaling Mediated EMT
PLOS ONE
IF0

