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Glucose neurotoxicity
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DOI：10.1038/nrn2294.png)
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Neurons have a constantly high glucose demand, and unlike muscle cells they cannot accommodate episodic glucose uptake under the influence of insulin. Neuronal glucose uptake depends on the extracellular concentration of glucose, and cellular damage can ensue after persistent episodes of hyperglycaemia - a phenomenon referred to as glucose neurotoxicity. This article reviews the pathophysiological manifestation of raised glucose in neurons and how this can explain the major components of diabetic neuropathy.
Keyword:
EXPERIMENTAL DIABETIC-NEUROPATHY
NERVE-CONDUCTION-VELOCITY
ACTIVATED PROTEIN-KINASE
ALDOSE REDUCTASE INHIBITOR
GLYCATION END-PRODUCTS
DORSAL-ROOT GANGLION
SUBCUTANEOUS INSULIN INFUSION
TANDEM MASS-SPECTROMETRY
AXO-GLIAL DYSJUNCTION
PERIPHERAL-NERVE
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