.png){kind=icon}
.png){kind=icon}
Return
Inhibition of ERO1A by TCF21 curbs aerobic glycolysis and enhances immune recognition in lung adenocarcinoma
.png){kind=icon}
PRE
AI
X
B
张
S
Y
DOI：10.1016/j.biocel.2025.106831.png)
.png){kind=icon}
Original request for help.png){kind=icon}
Share.png){kind=icon}
Save
Abstract
En 中文
Among the many malignancies, lung adenocarcinoma (LUAD) is a top global health and life risk. The focus of this research is to explore how the TCF21/ERO1A pathway influences the immune escape mechanisms triggered by aerobic glycolysis in LUAD.
Journal
I
IF:
2.8
Papers:
5.9K
Citations:
1.5W
Organization
No organization information available
Cited Papers