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Serial Changes in Epicardial Adipose Tissue Attenuation Identify Residual Atherosclerotic Risk Independent of LDL Lowering
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DOI：10.1016/j.jcmg.2026.05.014.png)
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Abstract
En 中文
Coronary plaque progression persists in some patients despite effective low-density lipoprotein cholesterol (LDL-C) lowering. Epicardial adipose tissue (EAT) is a metabolically active visceral fat depot that may influence coronary atherosclerosis through inflammatory signaling. The authors hypothesized that longitudinal changes in EAT would influence plaque progression.
Keywords:
adipose tissue attenuation
atherosclerosis
coronary computed tomography angiography
coronary plaque
epicardial adipose tissue
residual cardiovascular risk
AIC
Akaike information criterion
ANCOVA
analysis of covariance
CT
computed tomography
CTA
computed tomography angiography
EAT
epicardial adipose tissue
LDL-C
low-density lipoprotein cholesterol
NCP
noncalcified plaque
PAV
percent atheroma volume
PCAT
pericoronary adipose tissue
WC
waist circumference
Journal
J
IF:
0
Papers:
136
Citations:
0
Organization
Cited Papers