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Targeting the immunometabolism interface: A novel strategy for IPF therapy
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DOI:10.1016/j.pupt.2025.102394.png)
Abstract
En 中文
• Immunometabolism crosstalk drives IPF progression. • PPARγ and SPP1 are key regulatory genes in IPF. • Targeting Immunometabolism dysfunction offers new therapies. • Metabolic biomarkers aid in IPF diagnosis and treatment. • Future research should bridge basic and clinical IPF studies.
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2.1K
Citations:
2.6K
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