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The bile acid receptor TGR5 promotes functional recovery after spinal cord injury by rescuing mitochondrial dysfunction and suppressing AIM2-mediated pyroptosis
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DOI：10.1111/bph.70619.png)
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Abstract
En 中文
Previous studies have highlighted the significance of the bile acid receptor TGR5 (also known as Takeda G protein-coupled receptor 5) in regulating inflammation and mitochondrial homeostasis in various diseases, whereas the specific involvement of TGR5 in spinal cord injury (SCI) remains unclear. This study aimed to elucidate the effects of TGR5 on SCI, as well as the underlying mechanisms.
Keywords:
AIM2
mitochondria
spinal cord injury
TGR5
Journal
IF:
7.7
Papers:
1.4W
Citations:
3.7W
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