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AMPK-driven immunometabolic reprogramming enhances CD19+CD21hiCD24hi regulatory B cell-mediated suppression of atopic dermatitis
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DOI：10.1016/j.jaci.2026.06.015.png)
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Abstract
En 中文
Atopic dermatitis (AD) is characterized by dysregulated immune responses and persistent inflammation. Regulatory B cells (Bregs) suppress inflammation, but the metabolic mechanisms constraining their function in AD remain unclear.
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