.png){kind=icon}
.png){kind=icon}
Return
Amyloid exacerbates tau and alpha-synuclein pathologies, behavioral impairments, and neuroinflammation in a mixed dementia model
.png){kind=icon}
OA
AI
B
L
L
K
D
J
N
DOI：10.1002/alz.71639.png)
.png){kind=icon}
.png){kind=icon}
Share.png){kind=icon}
Save
Abstract
En 中文
Neurodegenerative diseases often involve overlapping alpha-synuclein (asyn), amyloid beta, and tau proteinopathies, yet the mechanisms, impact, and directionality of their interactions remain unclear.
Keywords:
adeno-associated virus (AAV)
A152T
amyloid beta
animal model
Alzheimer's disease
alpha-synuclein
E46K
LGALS3
Lewy body disease
Lewy body dementia
knock-in
mixed pathologies
neuroinflammation
Parkinson's disease
tau
AI Summary
Key information extracted from the uploaded paper, including a brief overview, abstract, background, key highlights, visual analysis, and future outlook.
Journal
A
IF:
0
Papers:
511
Citations:
0
Organization
View more organizations