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Bisphenol A exposure, stroke, and endothelial-related mechanisms: Association and mechanistic exploration
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DOI：10.1016/j.hazadv.2026.101422.png)
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Abstract
En 中文
• Higher urinary BPA exposure was associated with greater stroke prevalence. • Shared BPA-stroke targets converged on vascular and inflammatory pathways. • ESR1, NOS3, and F13A1 were prioritized as candidate BPA-stroke genes. • Single-cell analysis localized BPA-stroke signatures to activated endothelium. • BPA-treated HUVECs showed changes in endothelial dysfunction-related markers.
Keywords:
Environmental health
Endothelial dysfunction
Single-cell analysis
Bisphenol A
Molecular docking
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7.7
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1.4K
Citations:
3.1K
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