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Congenital Kir6.2 deficiency, but not acute KATP channel blockade, attenuates apomorphine-induced locomotor activity in mice
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DOI：10.1016/j.pbb.2026.174209.png)
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Abstract
En 中文
• Kir6.2−/− mice exhibited attenuated apomorphine-induced locomotor activation. • Acute KATP channel blockade did not reproduce the Kir6.2−/− phenotype. • Kir6.2 deficiency selectively impaired dopaminergic motor responsiveness. • Basal hedonic behavior was preserved in Kir6.2−/− mice.
Keywords:
Kir6.2 deficiency
KATP channel
dopaminergic motor responsiveness
apomorphine-induced locomotor activity
hedonic behavior
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