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Enhancing the anti-ageing capacity of hUC-MSCs via NMN co-treatment in D-galactose-induced ageing mice and cellular senescence models
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DOI：10.1016/j.mad.2026.112232.png)
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Abstract
En 中文
• Combined NMN and hUC-MSCs synergistically ameliorate immunosenescence and restore organ function in D-galactose-induced ageing mice. • The combined treatment suppresses lymphocyte senescence and restores redox balance. • NMN protects hUC-MSCs from microenvironment-induced senescence via activation of the Nrf2/HO-1 pathway.
Keywords:
hUC-MSCs
NMN
Immunosenescence
Oxidative stress
Nrf2/HO-1 pathway
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