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Exogenous mitochondrial transplantation attenuates oxidative stress–driven retinal degeneration in a sodium iodate – induced mouse model
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DOI：10.1016/j.neurobiolaging.2026.07.002.png)
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Abstract
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• Mitochondrial dysfunction promotes oxidative stress and inflammation in dry AMD. • Sodium iodate disrupts RPE mitochondria and barrier integrity. • Mitochondrial transfer modulates ROS–inflammasome and complement signaling. • Intravitreal mitochondrial therapy offers a cell-free strategy for dry AMD.
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