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Exploring the role of β2- and β3-adrenergic receptors in cystic fibrosis
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DOI：10.1016/j.pupt.2025.102385.png)
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Abstract
En 中文
Cystic fibrosis (CF) is an autosomal recessive disorder that affects multiple organs, with clinical manifestations, disease progression, and response to therapy varying among individuals. This effect is mainly caused by mutations in the gene encoding for the CF transmembrane conductance regulator (CFTR), a cAMP-regulated chloride channel.
Keywords:
cystic fibrosis
CFTR
chloride channel
autosomal recessive disorder
gene mutation
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