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Heat stress promotes myocardial injury and ventricular arrhythmia through DRP1-mediated mitochondrial dysfunction
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PRE
AI
J
H
J
S
N
H
张
黄
Y
H
M
J
C
Y
M
S
B
DOI：10.1016/j.freeradbiomed.2026.07.042.png)
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Abstract
En 中文
• Results systematically characterize myocardial injury and cardiac electrophysiological abnormalities caused by heat stroke. • Heat stress mediates mitochondrial dysfunction through the Ca2+/Calcineurin/p-DRP1(Ser637) signaling axis leading to myocardial injury and ventricular arrhythmias. • MitoTEMPO and FK506 ameliorates heat stroke-induced myocardial injury and ventricular arrhythmias.
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8.2
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2.1W
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5.4W
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