.png){kind=icon}
.png){kind=icon}
Return
Heme oxygenase/carbon monoxide in thermoregulation and neuroenergetics
.png){kind=icon}
OA
AI
E
L
DOI：10.1016/j.neuint.2026.106205.png)
.png){kind=icon}
.png){kind=icon}
Share.png){kind=icon}
Save
Abstract
En 中文
• HO/CO signaling modulates mitochondrial bioenergetics, redox balance, and thermoregulatory output under inflammatory stress • Thermoregulatory responses emerge from state-dependent integration of metabolic and autonomic control pathways • CO/HO effects are context-dependent, shaped by oxygen availability, timing, and inflammatory phenotype • Mitochondrial and redox adaptations provide a mechanistic link between immune activation and thermal phenotype • Integrated profiling of thermal, immune, and redox signals may improve targeting of HO/CO-based interventions
Keywords:
heme oxygenase-1
carbon monoxide
thermoregulation
neuroenergetics
mitochondria
neuroinflammation
sepsis
AI Summary
Key information extracted from the uploaded paper, including a brief overview, abstract, background, key highlights, visual analysis, and future outlook.
Journal
IF:
4
Papers:
5.3K
Citations:
1.0W
Organization
Cited Papers