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β-Hydroxybutyrate reduces neuronal excitability via GIRK channels
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DOI：10.1002/epi.70428.png)
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Abstract
En 中文
The ketogenic diet is used to treat drug-resistant epilepsy, yet the molecular mechanisms coupling metabolic state to seizure suppression remain understudied. β-Hydroxybutyrate (β-HB), a principal ketone body, exerts antiseizure effects, yet its downstream ionic mechanisms remain unclear.
Keywords:
ATP-sensitive potassium (KATP) channels
G protein-gated inwardly rectifying potassium (GIRK) channels
ketogenic diet
status epilepticus
β-hydroxybutyrate
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