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Lactylation of lysine396 in TNFRSF25 by lysine acetyltransferase 6B aggravates ferroptosis in metabolic dysfunction–associated steatohepatitis
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DOI：10.1111/bph.70517.png)
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Abstract
En 中文
Metabolic dysfunction–associated steatohepatitis (MASH) is a severe clinical complication of metabolic syndrome, but effective pharmacological treatments remain scarce. This study investigated the role of lactylation—a novel post-translational modification—in the pathogenesis of MASH, using murine models.
Keywords:
ferroptosis
Kat6B
lactylation
MASH
Tnfrsf25
Journal
IF:
7.7
Papers:
1.4W
Citations:
3.7W
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Cited Papers