.png){kind=icon}
.png){kind=icon}
Return
LXR/RXR activation mitigates ferroptosis in diabetic cardiomyocytes by regulating Calpain1 via Calpastatin
.png){kind=icon}
PRE
AI
J
F
H
X
K
T
Y
J
Q
DOI：10.1016/j.bcp.2026.118307.png)
.png){kind=icon}
Original request for help.png){kind=icon}
Share.png){kind=icon}
Save
Abstract
En 中文
Ferroptosis significantly contributes to myocardial injury in Type 2 Diabetes Mellitus (T2DM). Liver X Receptor (LXR) and Retinoid X Receptor (RXR) play crucial roles in lipid metabolism and inflammation, but their involvement in regulating ferroptosis in diabetic cardiomyocytes is not fully understood.
Journal
IF:
5.6
Papers:
1.3W
Citations:
3.3W
Organization
Cited Papers