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Melatonin maintains human umbilical vein endothelial cells function through potential regulation of the AKT-mTOR signaling pathway involving SIRT7 in preeclampsia
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DOI：10.1016/j.mce.2026.112825.png)
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Abstract
En 中文
• Serum melatonin levels were decreased in patients with preeclampsia. • Melatonin was demonstrated to ameliorate oxidative stress-induced dysfunction of HUVECs. • Melatonin significantly enhanced HUVEC function by upregulating SIRT7 expression and was associated with the AKT/mTOR signaling pathway.
Keywords:
melatonin
preeclampsia
HUVECs
SIRT7
AKT/mTOR signaling pathway
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