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Nuclear factor programming improves stem-cell-derived hepatocyte phenotype
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DOI：10.1016/j.stem.2022.04.009.png)
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Abstract
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In this issue of Cell Stem Cell, Ma et al. demonstrate that the activation of the nuclear receptor thyroid hormone receptor beta (NR1A2) improves the differentiation status of hepatocyte-like cells derived from human pluripotent stem cells.
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