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PM2.5 augments cigarette smoke-induced lung inflammation in mice by driving a stronger immune response: Potential beneficial effects of oleanolic acid
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DOI：10.1016/j.pupt.2025.102379.png)
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Abstract
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• PM2.5 accelerates and switches cigarette smoke-induced macrophagic inflammation to neutrophilic one. • Pro-inflammatory mediators and oxidative stress cause the CS+PM2.5-induced COPD pathogenesis. • Oleanolic acid reduces oxidative stress and brings protease-antiprotease balance to generate anti-inflammatory response. • Antioxidant and anti-inflammatory properties of oleanolic acid help in reversing lung damage marked by improved lung function.
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