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Smooth muscle cells transiently acquire a CD34+ progenitor state upon injury to drive pathological vascular remodeling
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DOI：10.1016/j.stem.2026.04.023.png)
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Abstract
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• Vascular injury induces a CD34+ transient progenitor state in SMCs, defined as STPCs • STPCs are highly proliferative and constitute the major source of neointimal SMCs • Genetic ablation of STPCs attenuates neointimal hyperplasia • Doublecortin-like kinase 1 (DCLK1) is an important regulator of STPC generation
Keywords:
STPCs
vascular injury
neointimal hyperplasia
CD34+ progenitor state
DCLK1
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