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Targeting soluble PD-1 alleviates peritoneal fibrosis by modulating PD-L1 recycling and mesothelial-mesenchymal transition
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DOI：10.1016/j.trsl.2026.07.005.png)
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Abstract
En 中文
Peritoneal fibrosis (PF) is a major cause of technique failure in long-term peritoneal dialysis (PD) patients, driven by a chronic microinflammatory state. While T-cell activation is implicated, the role of soluble programmed death-1 (sPD-1), primarily derived from activated T cells, in PF pathogenesis remains elusive.
Keywords:
sPD-1
Peritoneal fibrosis
Peritoneal dialysis
Mesothelial-mesenchymal transition
Endosomes
Targeted therapy
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