.png){kind=icon}
.png){kind=icon}
Return
The effect of dehydroepiandrosterone on the vascular system: role of H2S/NO pathways
.png){kind=icon}
OA
AI
C
C
M
E
A
V
R
E
N
DOI：10.1016/j.ejphar.2026.178957.png)
.png){kind=icon}
.png){kind=icon}
Share.png){kind=icon}
Save
Abstract
En 中文
• Hydrogen sulfide, together with nitric oxide, is involved in DHEA-induced relaxation in mouse aorta • DHEA exposure causes an increase in hydrogen sulfide in BAEC • DHEA causes vasorelaxation either in the presence or in the absence of endothelium • DHEA vasorelaxant effect involved KATP and potassium and L-type calcium channels • DHEA vasorelaxing effect does not involve estrogen and androgen receptors activation.
Keywords:
dehydroepiandrosterone
relaxation
hydrogen sulfide
nitric oxide
mice aorta
BAEC
AI Summary
Key information extracted from the uploaded paper, including a brief overview, abstract, background, key highlights, visual analysis, and future outlook.
Journal
IF:
4.7
Papers:
2.3W
Citations:
4.0W
Organization
Cited Papers