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The high-carbohydrate diet delayed skin wound healing through suppressing AGEs-MTOR-HIF1A-driven glycolysis in turbot, Scophthalmus maximus L.
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DOI：10.1016/j.jnutbio.2026.110436.png)
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Abstract
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• First evidence that the high-carbohydrate (HC) diet impair skin wound healing inglucose-intolerant turbot. • The HC diet induced hyperglycemia/hyperinsulinemia and delayed wound closure. • The HC diet led to suppression of early re-epithelialization, aberrant inflammation,deficient provisional matrix deposition, collagen production, and angiogenesis. • The HC diet delayed skin wound healing by the suppression of AGEs-MTORHIF1A-driven glycolysis.
Keywords:
High-carbohydrate diet
Skin wound healing
AGEs
MTOR
HIF1A
Turbot
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