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The SMYD3-MEK/ERK Axis Contributes to Intestinal Metaplasia and H. pylori-associated Gastric Carcinogenesis
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DOI：10.1016/j.labinv.2026.106137.png)
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Abstract
En 中文
Chronic Helicobacter pylori (H. pylori) infection induces gastric atrophy and intestinal metaplasia (IM), which are key precancerous lesions for gastric cancer (GC). The purpose of this study was to determine the mechanisms by which MEK/ERK signaling persists from active infection through the post-eradication stage and contributes to gastric carcinogenic risk.
Keywords:
SMYD3
MEK/ERK signaling
Helicobacter pylori
intestinal metaplasia
gastric carcinogenesis
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