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Thymoquinone enhances kidney function in obesity-induced renal impairment by targeting redox balance, inflammation, autophagy, and apoptosis
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DOI：10.1016/j.tice.2026.103568.png)
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Abstract
En 中文
• Thymoquinone (TQ) reverses HFD-induced kidney dysfunction (creatinine, urea, uric acid) and tubular damage (KIM-1, NGAL). • TQ restores podocyte integrity, evidenced by increased podocin and nephrin levels. • TQ orchestrates a novel regulation of autophagy flux and mitigates DNA damage in renal tissue. • TQ inhibits mitochondrial apoptosis, providing a multi-pathway defense against obesity-related nephropathy.
Keywords:
Thymoquinone
kidney function
autophagy
apoptosis
inflammation
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