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Transient ocular hypertension drives delayed and IOP-independent retinal neurodegeneration
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DOI：10.1016/j.nbd.2026.107540.png)
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Abstract
En 中文
• Neurodegeneration persists despite the reduction of intraocular pressure (IOP). • Longitudinal tracking in a reversible OHT model reveals neurodegenerative pathology. • IOP normalization partially rescues early function but cannot completely prevent delayed decline. • IOP-independent neurodegeneration persists after primary mechanical stress relief.
Keywords:
glaucoma
Reversible ocular hypertension
Delayed neurodegeneration
Retinal ganglion cells
Neuroprotection
IOP-independent
Longitudinal tracking
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