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Traumatic brain injury enhances fracture healing by upregulating VNN1 to activate the Wnt/β-catenin signaling pathway
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DOI:10.1016/j.gene.2026.150291.png)
Abstract
En 中文
• VNN1 is identified as a key gene mediating TBI-accelerated fracture healing. • VNN1 expression peaks earlier and lasts longer in fractures with TBI. • This study provides a novel molecular target for treating delayed fracture healing.
Keywords:
Traumatic brain injury
Fracture
VNN1
Wnt/β-catenin
Differentially expressed genes

