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VBIT-4 attenuates diabetes-associated cardiac mitochondrial dysfunction and oxidative stress in a mouse model of diabetes
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DOI：10.1016/j.freeradbiomed.2026.07.049.png)
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Abstract
En 中文
• VBIT-4 lowers blood glucose levels in diabetic mice. • VBIT-4 reduces MDA levels in cardiac mitochondria of diabetic animals. • Pharmacological and genetic modulation of VDAC1 reduces DCF fluorescence in cells under hyperlipidemic conditions. • VBIT-4 interacts with the α-helical N-terminus of VDAC1, stabilizing it within the channel pore
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