Minfeng Shu

fudan university

H指数:21

被引数:1,228

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An IGF2BP3-dependent metabolic circuit governs macrophage recruitment and immunosuppression in glioblastoma

IGF2BP3依赖性代谢回路调控胶质母细胞瘤中巨噬细胞的募集和免疫抑制

Cell Reports

IF6.9

2026-07-030

OAAI

A novel risk model incorporating MMP9, TLR8, and LILRB2 drives neutrophil extracellular trap formation and promotes immune evasion in glioma

一种整合MMP9、TLR8和LILRB2的新型风险模型驱动中性粒细胞胞外陷阱形成并促进胶质瘤中的免疫逃逸

International Immunopharmacology

IF4.7

2026-02-240

PRE

HSV-1-induced N6-methyladenosine reprogramming via ICP0-mediated suppression of METTL14 potentiates oncolytic activity in glioma

CELL REPORTS

IF6.9

2024-10-011

OAAI

NSUN5/TET2-directed chromatin- associated RNA modification of 5-methylcytosine to 5-hydroxymethylcytosine governs glioma immune evasion

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

IF9.1

2024-03-2810

PRE

Sublethal heat treatment enhances lactic acid uptake in macrophages via MCT1, leading to reduced paraspeckle formation and a subsequent decrease in macrophage pyroptosis

FRONTIERS IN IMMUNOLOGY

IF5.9

2024-01-111

OAAI

Overcoming therapeutic resistance in oncolytic herpes virotherapy by targeting IGF2BP3-induced NETosis in malignant glioma

通过靶向恶性神经胶质瘤中的IGF2BP3-induced NETosis克服溶瘤性疱疹病毒治疗中的治疗抗性

NATURE COMMUNICATIONS

IF15.7

2024-01-0213

OAAI

IDH1 mutation impairs antiviral response and potentiates oncolytic virotherapy in glioma

NATURE COMMUNICATIONS

IF15.7

2023-10-256

OAAI

Therapeutic targeting m6A-guided miR-146a-5p signaling contributes to the melittin-induced selective suppression of bladder cancer

CANCER LETTERS

IF10.1

2022-05-0136

PRE

Circadian Regulation Patterns With Distinct Immune Landscapes in Gliomas Aid in the Development of a Risk Model to Predict Prognosis and Therapeutic Response

FRONTIERS IN IMMUNOLOGY

IF5.9

2022-01-068

OAAI

miR-221/222 suppression induced by activation of the cAMP/PKA/CREB1 pathway is required for cAMP-induced bidirectional differentiation of glioma cells

FEBS LETTERS

IF3

2021-10-273

Role of activating transcription factor 3 in the synthesis of latency-associated transcript and maintenance of herpes simplex virus 1 in latent state in ganglia

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

IF9.1

2015-08-2427

OAAI

Transcriptional upregulation of microtubule-associated protein 2 is involved in the protein kinase A-induced decrease in the invasiveness of glioma cells

NEURO-ONCOLOGY

IF13.4

2015-05-2525

OAAI

Tristetraprolin Recruits the Herpes Simplex Virion Host Shutoff RNase to AU-Rich Elements in Stress Response mRNAs To Enable Their Cleavage

JOURNAL OF VIROLOGY

IF3.8

2015-05-1526

OAAI

Cholera toxin, a typical protein kinase A activator, induces G1 phase growth arrest in human bladder transitional cell carcinoma cells via inhibiting the c-Raf/MEK/ERK signaling pathway

霍乱毒素是一种典型的蛋白激酶a激活剂，通过抑制c-raf/MEK/ERK信号通路诱导人膀胱移行细胞癌细胞G1期生长停滞

MOLECULAR MEDICINE REPORTS

IF3.5

2014-03-1415

OAAI

The Nuclear-Cytoplasmic Shuttling of Virion Host Shutoff RNase Is Enabled by pUL47 and an Embedded Nuclear Export Signal and Defines the Sites of Degradation of AU-Rich and Stable Cellular mRNAs

JOURNAL OF VIROLOGY

IF3.8

2013-12-1525

OAAI

Selective degradation of mRNAs by the HSV host shutoff RNase is regulated by the UL47 tegument protein

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

IF9.1

2013-04-1564

OAAI

MicroRNA 335 Is Required for Differentiation of Malignant Glioma Cells Induced by Activation of cAMP/Protein Kinase A Pathway

MOLECULAR PHARMACOLOGY

IF3

2012-03-0148

PREAI

MiR-135a functions as a selective killer of malignant glioma

ONCOGENE

IF7.3

2011-12-0584

PREAI

Targeting oncogenic miR-335 inhibits growth and invasion of malignant astrocytoma cells

MOLECULAR CANCER

IF33.9

2011-05-19123

OAAI

Activation of a pro-survival pathway IL-6/JAK2/STAT3 contributes to glial fibrillary acidic protein induction during the cholera toxin-induced differentiation of C6 malignant glioma cells

在霍乱毒素诱导的C6恶性神经胶质瘤细胞分化过程中，促生存途径IL-6/JAK2/STAT3的激活有助于胶质纤维酸性蛋白的诱导

MOLECULAR ONCOLOGY

IF4.5

2011-03-2129

OAAI

Minfeng Shu

个人简介

教育背景

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