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收藏Disruption of the EGFR-SQSTM1 interaction by a stapled peptide suppresses lung cancer via activating autophagy and inhibiting EGFR signaling( vol 474 , pg 23 , 2020)通过一种 Stapled 肽破坏 EGFR-SQSTM1 相互作用,能够通过激活自噬和抑制 EGFR 信号通路来抑制肺癌(卷 474,页 23,2020)。
Yu, Jiao-jiao; Zhou, Dan-dan; Cui, Bing; Zhang, Cheng; Tan, Feng-wei; Chang, Shan; Li, Ke; Lv, Xiao-xi; Zhang, Xiao-wei; Shang, Shuang; Xiang, Yu-Jin; Chen, Fei; Yu, Jin-mei; Liu, Shan-shan; Wang, Feng; Hu, Zhuo-Wei; Hua, Fang
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收藏TRIB3 promotes pulmonary fibrosis through inhibiting SLUG degradation by physically interacting with MDM2
Lv, Xiaoxi; Liu, Shanshan; Liu, Chang; Li, Yunxuan; Zhang, Tingting; Qi, Jie; Li, Ke; Hua, Fang; Cui, Bing; Zhang, Xiaowei; Liu, Yuxin; Yu, Jiaojiao; Yu, Jinmei; Li, Li; Li, Xia; Yao, Zhigang; Huang, Bo
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收藏Comprehensive RNA-seq reveals molecular changes in kidney malignancy among people living with HIV
Bao, Juan; Ye, Jianqing; Xu, Jingjing; Liu, Shanshan; Wang, Lin; Li, Zehuan; Li, Qiuyue; Liu, Feng; He, Xiaomeng; Zou, Heng; Feng, Yanling; Corpe, Christopher; Zhang, Xiaoyan; Xu, Jianqing; Zhu, Tongyu; Wang, Jin
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收藏The cell cycle inhibitor P21 promotes the development of pulmonary fibrosis by suppressing lung alveolar regeneration
Lv, Xiaoxi; Liu, Chang; Liu, Shanshan; Li, Yunxuan; Wang, Wanyu; Li, Ke; Hua, Fang; Cui, Bing; Zhang, Xiaowei; Yu, Jiaojiao; Yu, Jinmei; Hu, ZhuoWei
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收藏The chemokine CCL1 triggers an AMFR-SPRY1 pathway that promotes differentiation of lung fibroblasts into myofibroblasts and drives pulmonary fibrosis (vol 54, pg 2042, 2021)
Liu, Shan-shan; Liu, Chang; Lv, Xiao-xi; Cui, Bing; Yan, Jun; Li, Yun-xuan; Li, Ke; Hua, Fang; Zhang, Xiao-wei; Yu, Jiao-jiao; Yu, Jin-mei; Wang, Feng; Shang, Shuang; Li, Ping-ping; Zhou, Zhi-guang; Xiao, Yang; Hu, Zhuo-wei
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收藏TRIB3-GSK-3β interaction promotes lung fibrosis and serves as a potential therapeutic targetTRIB3-GSK-3β 相互作用促进肺纤维化并作为潜在的治疗靶点
Liu, Shanshan; Lv, Xiaoxi; Wei, Xupeng; Liu, Chang; Li, Qiao; Min, Jiali; Hua, Fang; Zhang, Xiaowei; Li, Ke; Li, Pingping; Xiao, Yang; Hu, Zhuowei; Cui, Bing
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收藏The chemokine CCL1 triggers an AMFR-SPRY1 pathway that promotes differentiation of lung fibroblasts into myofibroblasts and drives pulmonary fibrosis
Liu, Shan-Shan; Liu, Chang; Lv, Xiao-Xi; Cui, Bing; Yan, Jun; Li, Yun-Xuan; Li, Ke; Hua, Fang; Zhang, Xiao-Wei; Yu, Jiao-Jiao; Yu, Jin-Mei; Wang, Feng; Shang, Shuang; Li, Ping-Ping; Zhou, Zhi-Guang; Xiao, Yang; Hu, Zhuo-Wei
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收藏Faciogenital Dysplasia 5 supports cancer stem cell traits in basal-like breast cancer by enhancing EGFR stability
Li, Ke; Zhang, Ting-ting; Zhao, Chen-xi; Wang, Feng; Cui, Bing; Yang, Zhao-na; Lv, Xiao-xi; Yeerjiang, Zaiwuli; Yuan, Yu-fen; Yu, Jin-mei; Wang, Zhen-he; Zhang, Xiao-wei; Yu, Jiao-jiao; Liu, Shan-shan; Shang, Shuang; Huang, Bo; Hua, Fang; Hu, Zhuo-wei
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收藏TRIB3 promotes MYC-associated lymphoma development through suppression of UBE3B-mediated MYC degradation
Li, Ke; Wang, Feng; Yang, Zhao-na; Zhang, Ting-ting; Yuan, Yu-fen; Zhao, Chen-xi; Yeerjiang, Zaiwuli; Cui, Bing; Hua, Fang; Lv, Xiao-xi; Zhang, Xiao-wei; Yu, Jiao-jiao; Liu, Shan-shan; Yu, Jin-mei; Shang, Shuang; Xiao, Yang; Hu, Zhuo-wei
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收藏TRIB3-EGFR interaction promotes lung cancer progression and defines a therapeutic target
Yu, Jiao-jiao; Zhou, Dan-dan; Yang, Xiao-xiao; Cui, Bing; Tan, Feng-wei; Wang, Junjian; Li, Ke; Shang, Shuang; Zhang, Cheng; Lv, Xiao-xi; Zhang, Xiao-wei; Liu, Shan-shan; Yu, Jin-mei; Wang, Feng; Huang, Bo; Hua, Fang; Hu, Zhuo-Wei
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收藏MLH1 Deficiency Induces Cetuximab Resistance in Colon Cancer via Her-2/PI3K/AKT Signaling
Han, Ying; Peng, Yinghui; Fu, Yaojie; Cai, Changjing; Guo, Cao; Liu, Shanshan; Li, Yiyi; Chen, Yihong; Shen, Edward; Long, Kexin; Wang, Xinwen; Yu, Jian; Shen, Hong; Zeng, Shan
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收藏Disruption of the EGFR-SQSTM1 interaction by a stapled peptide suppresses lung cancer via activating autophagy and inhibiting EGFR signaling
Yu, Jiao-jiao; Zhou, Dan-dan; Cui, Bing; Zhang, Cheng; Tan, Feng-wei; Chang, Shan; Li, Ke; Lv, Xiao-xi; Zhang, Xiao-wei; Shang, Shuang; Xiang, Yu-Jin; Chen, Fei; Yu, Jin-mei; Liu, Shan-shan; Wang, Feng; Hu, Zhuo-Wei; Hua, Fang
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