Anti-RANKL improves muscle dysfunction and strengthens bone in laminin-α2-deficient dy2J/dy2J dystrophic mice.抗-RANKL改善层粘连蛋白-α2缺乏的dy2J/dy2J肌营养不良小鼠的肌肉功能障碍并增强骨骼the american journal of pathologyIF02026-05-110OAAI
A 12-Week Strength Training Improves Mitochondrial Respiration, H2O2 Emission and Skeletal Muscle Integrity in Women With Myotonic Dystrophy Type 1ACTA PHYSIOLOGICAIF5.62025-12-010OAAI
Morphological and functional phenotyping of skeletal muscle and bone in the zQ175 knock-in mouse model of Huntington's diseaseJOURNAL OF HUNTINGTONS DISEASEIF3.12025-10-010PRE
Anti-RANKL Therapy Prevents Glucocorticoid-Induced Bone Loss and Promotes Muscle Function in a Mouse Model of Duchenne Muscular DystrophyCALCIFIED TISSUE INTERNATIONALIF3.22023-07-209OAAI
RANKL Inhibition Reduces Cardiac Hypertrophy in mdx Mice and Possibly in Children with Duchenne Muscular DystrophyCELLSIF5.22023-06-037OAAI
The inflammatory response, a mixed blessing for muscle homeostasis and plasticityFRONTIERS IN PHYSIOLOGYIF3.42022-11-233OAAI
Muscle weakness and selective muscle atrophy in osteoprotegerin-deficient miceHUMAN MOLECULAR GENETICSIF3.22020-01-1523OAAI
An anti-RANKL treatment reduces muscle inflammation and dysfunction and strengthens bone in dystrophic miceHUMAN MOLECULAR GENETICSIF3.22019-06-1053PREAI
Targeting the Muscle-Bone Unit: Filling Two Needs with One Deed in the Treatment of Duchenne Muscular Dystrophycurrent pollution reportsIF5.32018-09-1728PREAI
Genetic deletion of muscle RANK or selective inhibition of RANKL is not as effective as full-length OPG-fc in mitigating muscular dystrophyACTA NEUROPATHOLOGICA COMMUNICATIONSIF5.72018-04-2450OAAI
Selectins Ligand Decorated Drug Carriers for Activated Endothelial Cell TargetingBIOCONJUGATE CHEMISTRYIF3.92008-09-2031PREAI