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Yeye Kuang

zhe jiang university

H指数:12

被引数:423

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FOXS1, frequently inactivated by promoter methylation, inhibited colorectal cancer cell growth by promoting TGFBI degradation through autophagy-lysosome pathway

FOXS1常因启动子甲基化而失活，可通过促进TGFBI经自噬-溶酶体途径降解来抑制结直肠癌细胞生长。

Journal of Advanced Research

IF13

2025-01-240

OAAI

Progress of research on γδ T cells in colorectal cancer (Review)

ONCOLOGY REPORTS

IF3.9

2024-10-041

PRE

Forkhead box E1, frequently downregulted by promoter methylation, inhibits colorectal cancer cell growth and migration

CANCER CELL INTERNATIONAL

IF6

2024-05-111

OAAI

IRS1 promotes thyroid cancer metastasis through EMT and PI3K/AKT pathways

CLINICAL ENDOCRINOLOGY

IF2.4

2024-01-033

PRE

FOXD3, frequently methylated in colorectal cancer, acts as a tumor suppressor and induces tumor cell apoptosis under ER stress via p53

FOXD3在结直肠癌中经常甲基化，可作为肿瘤抑制因子，并通过p53在ER应激下诱导肿瘤细胞凋亡。

IF2.9

2019-11-3019

PREAI

CHAC2, downregulated in gastric and colorectal cancers, acted as a tumor suppressor inducing apoptosis and autophagy through unfolded protein response

CHAC2在胃癌和结直肠癌中表达下调，作为肿瘤抑制因子通过未折叠蛋白反应诱导细胞凋亡和自噬

CELL DEATH & DISEASE

IF9.6

2017-08-2423

OAAI

BCLB, methylated in hepatocellular carcinoma, is a starvation stress sensor that induces apoptosis and autophagy through the AMPK-mTOR signaling cascade

IF10.1

2017-06-0140

PREAI

Epigenomic and Functional Characterization of Junctophilin 3 (JPH3) as a Novel Tumor Suppressor Being Frequently Inactivated by Promoter CpG Methylation in Digestive Cancers

IF13.3

2017-01-0114

OAAI

Protocadherin 17 acts as a tumour suppressor inducing tumour cell apoptosis and autophagy, and is frequently methylated in gastric and colorectal cancers

原钙粘蛋白17作为一种肿瘤抑制因子，诱导肿瘤细胞凋亡和自噬，在胃癌和结直肠癌中经常被甲基化

JOURNAL OF PATHOLOGY

IF5.2

2012-12-17100

PREAI

A novel variant of the RON receptor tyrosine kinase derived from colorectal carcinoma cells which lacks tyrosine phosphorylation but induces cell migration

来自大肠癌细胞的RON受体酪氨酸激酶的新变体，其缺乏酪氨酸磷酸化但诱导细胞迁移

EXPERIMENTAL CELL RESEARCH

IF3.5

2012-12-016

PREAI