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T

Tang, Jiping

loma linda university

H指数:0

被引数:3

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Inhibition of EZH2 (Enhancer of Zeste Homolog 2) Attenuates Neuroinflammation via H3k27me3/SOCS3/TRAF6/NF-κB (Trimethylation of Histone 3 Lysine 27/Suppressor of Cytokine Signaling 3/Tumor Necrosis Factor Receptor Family 6/Nuclear Factor-κB) in a Rat Model of Subarachnoid Hemorrhage (vol 51, pg 3320, 2020)

IF8.9

2024-07-010

PRE

AVE 0991 attenuates oxidative stress and neuronal apoptosis via Mas/PKA/CREB/UCP-2 pathway after subarachnoid hemorrhage in rats (vol 20, pg 75 , 2019)

IF11.9

2024-05-010

OA

LRP1 activation attenuates white matter injury by modulating microglial polarization through Shc1/PI3K/Akt pathway after subarachnoid hemorrhage in rats (vol 21, 101121, 2019)

IF11.9

2024-05-010

OA

Activation of galanin receptor 1 with M617 attenuates neuronal apoptosis via ERK/GSK-3β/TIP60 pathway after subarachnoid hemorrhage in rats (vol 18, pg 1905, 2021)

NEUROTHERAPEUTICS

IF6.9

2024-04-010

OA

Kynurenine/Aryl Hydrocarbon Receptor Modulates Mitochondria-Mediated Oxidative Stress and Neuronal Apoptosis in Experimental Intracerebral Hemorrhage

犬尿氨酸/芳香烃受体调节实验性脑出血中线粒体介导的氧化应激和神经元凋亡

ANTIOXIDANTS & REDOX SIGNALING

IF6.1

2022-12-0115

OA

A new perspective on cerebrospinal fluid dynamics after subarachnoid hemorrhage: From normal physiology to pathophysiological changes

JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM

IF4.5

2021-11-2238

OA

LRP1 activation attenuates white matter injury by modulating microglial polarization through Shc1/PI3K/Akt pathway after subarachnoid hemorrhage in rats

IF11.9

2019-02-01115

OAAI

AVE 0991 attenuates oxidative stress and neuronal apoptosis via Mas/PKA/CREB/UCP-2 pathway after subarachnoid hemorrhage in rats

IF11.9

2019-01-01138

OAAI

Controversies and evolving new mechanisms in subarachnoid hemorrhage

PROGRESS IN NEUROBIOLOGY

IF6.1

2014-04-01315

OAAI

Pontine hemorrhage RESPONSE

JOURNAL OF NEUROSURGERY

IF3.6

2013-06-010

PREAI

The hyperbaric oxygen preconditioning-induced brain protection is mediated by a reduction of early apoptosis after transient global cerebral ischemia

NEUROBIOLOGY OF DISEASE

IF5.6

2008-01-0198

OAAI