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Laura Tuohey

University of Melbourne

H指数:19

被引数:1,274

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Nicotinamide and its effects on endothelial dysfunction and secretion of antiangiogenic factors by primary human placental cells and tissues

IF2.5

2021-06-012

PREAI

Combining metformin and sulfasalazine additively reduces the secretion of antiangiogenic factors from the placenta: Implications for the treatment of preeclampsia

IF2.5

2020-06-0116

PREAI

Sulfasalazine decreases soluble fms-like tyrosine kinase-1 secretion potentially via inhibition of upstream placental epidermal growth factor receptor signalling

IF2.5

2019-11-0111

PREAI

Sulfasalazine reduces placental secretion of antiangiogenic factors, up-regulates the secretion of placental growth factor and rescues endothelial dysfunction

IF10.8

2019-03-0142

OAAI

Activating Transcription Factor 3 Is Reduced in Preeclamptic Placentas and Negatively Regulates sFlt-1 (Soluble fms-Like Tyrosine Kinase 1), Soluble Endoglin, and Proinflammatory Cytokines in Placenta

IF8.2

2017-11-0125

OAAI

Proton Pump Inhibitors Decrease Soluble fms-Like Tyrosine Kinase-1 and Soluble Endoglin Secretion, Decrease Hypertension, and Rescue Endothelial Dysfunction

IF8.2

2017-03-01129

OAAI

Key players of the necroptosis pathway RIPK1 and SIRT2 are altered in placenta from preeclampsia and fetal growth restriction

IF2.5

2017-03-0131

PREAI

Identifying late-onset fetal growth restriction by measuring circulating placental RNA in the maternal blood at 28 weeks' gestation

AMERICAN JOURNAL OF OBSTETRICS AND GYNECOLOGY

IF8.4

2016-04-0127

PREAI

Metformin as a prevention and treatment for preeclampsia: effects on soluble fms-like tyrosine kinase 1 and soluble endoglin secretion and endothelial dysfunction

AMERICAN JOURNAL OF OBSTETRICS AND GYNECOLOGY

IF8.4

2016-03-01168

OAAI

Jumonji Domain Containing Protein 6 Is Decreased in Human Preeclamptic Placentas and Regulates sFLT-1 Splice Variant Production

BIOLOGY OF REPRODUCTION

IF3

2016-03-0121

PREAI

Prediction of late onset fetal growth restriction using circulating placental RNA in the maternal blood

AMERICAN JOURNAL OF OBSTETRICS AND GYNECOLOGY

IF8.4

2016-01-010

OAAI

Placental-Specific sFLT-1 e15a Protein Is Increased in Preeclampsia, Antagonizes Vascular Endothelial Growth Factor Signaling, and Has Antiangiogenic Activity

胎盘特异性sFLT-1 e15a蛋白在先兆子痫中增加，拮抗血管内皮生长因子信号传导，并具有抗血管生成活性

IF8.2

2015-12-0174

OAAI

Heme Oxygenase-1 Is Not Decreased in Preeclamptic Placenta and Does Not Negatively Regulate Placental Soluble fms-Like Tyrosine Kinase-1 or Soluble Endoglin Secretion

IF8.2

2015-11-0132

OAAI

ACTIVATING TRANSCRIPTION FACTOR 3-A KEY MOLECULE IN THE PATHOGENESIS OF PREECLAMPSIA

IF2.5

2015-09-010

PREAI

PROTON PUMP INHIBITORS QUENCH THE PATHOPHYSIOLOGICAL CHARACTERISTICS OF PREECLAMPSIA IN BOTH HUMAN AND MOUSE MODELS AND REPRESENT AN EXCITING NOVEL CANDIDATE THERAPEUTIC

IF2.5

2015-09-010

PREAI

YC-1 reduces placental sFlt-1 and soluble endoglin production and decreases endothelial dysfunction: A possible therapeutic for preeclampsia

MOLECULAR AND CELLULAR ENDOCRINOLOGY

IF3.6

2015-09-0129

PREAI

Sofalcone Upregulates the Nuclear Factor (Erythroid-Derived 2)-Like 2/Heme Oxygenase-1 Pathway, Reduces Soluble fms-Like Tyrosine Kinase-1, and Quenches Endothelial Dysfunction Potential Therapeutic for Preeclampsia

IF8.2

2015-04-0146

PREAI

Activated Notch counteracts Ikaros tumor suppression in mouse and human T-cell acute lymphoblastic leukemia

IF13.4

2015-02-0631

OAAI

HO-1 EXPRESSION IS NOT REDUCED IN PREECLAMPTIC PLACENTA, AND DOES NOT REGULATE sFLT-1 OR SOLUBLE ENDOGLIN SECRETION IN EITHER PRIMARY TROPHOBLAST OR ENDOTHELIAL CELLS

IF2.5

2014-09-010

PREAI

PROTON PUMP INHIBITORS SIGNIFICANTLY QUENCH PATHOPHYSIO LOGICAL CHARACTERISTICS OF PREECLAMPSIA IN PRIMARY PLACENTAL AND ENDOTHELIAL MODELS: A NOVEL CANDIDATE THERAPEUTIC

IF2.5

2014-09-010

PREAI