Ha-Na Oh

Korea Institute of Toxicology

H指数:22

被引数:1,278

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Licochalcone D reduces H2O2-induced SH-SY5Y cell neurotoxicity by regulating reactive oxygen species

Licochalcone D通过调控活性氧（ROS）来减轻H2O2诱导的SH-SY5Y细胞神经毒性。

Frontiers in Pharmacology

IF4.8

2025-09-210

OAAI

Mechanistic insights into the neurotoxicity of Isothiazolinone biocides in human neuronal cells

Journal of Environmental Sciences

IF6.3

2025-09-190

OAAI

Assessment of poly (hexamethylenebicyanoguanide-hexamethylenediamine) hydrochloride-induced developmental neurotoxicity via oxidative stress mechanism: Integrative approaches with neuronal cells and zebrafish

JOURNAL OF HAZARDOUS MATERIALS

IF11.3

2024-03-013

OAAI

Human coculture model of astrocytes and SH-SY5Y cells to test the neurotoxicity of chemicals

ECOTOXICOLOGY AND ENVIRONMENTAL SAFETY

IF6.1

2024-01-013

OAAI

Copper pyrithione and zinc pyrithione induce cytotoxicity and neurotoxicity in neuronal/astrocytic co-cultured cells via oxidative stress

SCIENTIFIC REPORTS

IF3.9

2023-12-277

OAAI

Developmental neurotoxicity induced by glutaraldehyde in neuron/astrocyte co-cultured cells and zebrafish

戊二醛对神经元/星形胶质细胞共培养细胞和斑马鱼的发育神经毒性

ECOTOXICOLOGY AND ENVIRONMENTAL SAFETY

IF6.1

2022-09-0114

PRE

Targeted inhibition of c-MET by podophyllotoxin promotes caspase-dependent apoptosis and suppresses cell growth in gefitinib-resistant non-small cell lung cancer cells

鬼臼毒素靶向抑制c-met促进caspase依赖的细胞凋亡并抑制吉非替尼耐药非小细胞肺癌细胞的生长

PHYTOMEDICINE

IF8.3

2021-01-0122

PREAI

Janus kinase 2 inhibition by Licochalcone B suppresses esophageal squamous cell carcinoma growth

Licochalcone B抑制Janus激酶2抑制食管鳞状细胞癌的生长

PHYTOTHERAPY RESEARCH

IF6.3

2020-03-0620

PREAI

Retrochalcone Echinatin Triggers Apoptosis of Esophageal Squamous Cell Carcinoma via ROS- and ER Stress-Mediated Signaling Pathways

逆转查尔酮棘霉素通过ROS和ER应激介导的信号通路触发食管鳞状细胞癌的凋亡

MOLECULES

IF4.6

2019-11-0934

OAAI

Dual inhibition of EGFR and MET by Echinatin retards cell growth and induces apoptosis of lung cancer cells sensitive or resistant to gefitinib

PHYTOTHERAPY RESEARCH

IF6.3

2019-11-0723

PREAI

Licochalcone B inhibits growth and induces apoptosis of human non-small-cell lung cancer cells by dual targeting of EGFR and MET

Licochalcone B通过EGFR和MET的双重靶向抑制人非小细胞肺癌细胞的生长并诱导其凋亡

PHYTOMEDICINE

IF8.3

2019-10-0139

PREAI

Tyrosinase Inhibition Antioxidant Effect and Cytotoxicity Studies of the Extracts of Cudrania tricuspidata Fruit Standardized in Chlorogenic Acid

MOLECULES

IF4.6

2019-09-0719

OAAI

JAK2 regulation by licochalcone H inhibits the cell growth and induces apoptosis in oral squamous cell carcinoma

licochalcone H调节JAK2抑制口腔鳞状细胞癌细胞生长并诱导凋亡

PHYTOMEDICINE

IF8.3

2019-01-0129

PREAI

Licochalcone H induces the apoptosis of human oral squamous cell carcinoma cells via regulation of matrin 3

ONCOLOGY REPORTS

IF3.9

2018-10-1125

OAAI

Licochalcone C induced apoptosis in human oral squamous cell carcinoma cells by regulation of the JAK2/STAT3 signaling pathway

Licochalcone C通过调节JAK2/STAT3信号通路诱导人口腔鳞状细胞癌细胞凋亡

JOURNAL OF CELLULAR BIOCHEMISTRY

IF2.8

2018-08-2033

PREAI

Natural Compound Licochalcone B Induced Extrinsic and Intrinsic Apoptosis in Human Skin Melanoma (A375) and Squamous Cell Carcinoma (A431) Cells

天然化合物Licochalcone B诱导人皮肤黑色素瘤 (A375) 和鳞状细胞癌 (A431) 细胞的外在和内在凋亡

PHYTOTHERAPY RESEARCH

IF6.3

2017-10-1352

PREAI

Licochalcone A Suppresses Specificity Protein 1 as a Novel Target in Human Breast Cancer Cells

JOURNAL OF CELLULAR BIOCHEMISTRY

IF2.8

2017-06-1240

PREAI

Manumycin A induces apoptosis in malignant pleural mesothelioma through regulation of Sp1 and activation of the mitochondria-related apoptotic pathway

ONCOLOGY REPORTS

IF3.9

2016-05-108

OAAI

Caffeic acid phenethyl ester lessens disease symptoms in an experimental autoimmune uveoretinitis mouse model

EXPERIMENTAL EYE RESEARCH

IF2.7

2015-05-0113

PREAI

Rutin from Dendropanax morbifera Leveille Protects Human Dopaminergic Cells Against Rotenone Induced Cell Injury Through Inhibiting JNK and p38 MAPK Signaling

NEUROCHEMICAL RESEARCH

IF3.8

2014-02-1985

PREAI

Ha-Na Oh

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