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Optimized bisubstrate inhibitors for the actin N-terminal acetyltransferase NAA80FRONTIERS IN CHEMISTRYIF4.22023-06-200OAAI
Inhibition of VMAT2 by β2-adrenergic agonists, antagonists, and the atypical antipsychotic ziprasidoneCOMMUNICATIONS BIOLOGYIF5.12022-11-2310OAAI
A scavenger receptor B (CD36)-like protein is a potential mediator of intestinal heme absorption in the hematophagous ectoparasite Lepeophtheirus salmonisSCIENTIFIC REPORTSIF3.92019-03-1214OAAI
A novel NAA10 variant with impaired acetyltransferase activity causes developmental delay, intellectual disability, and hypertrophic cardiomyopathyEUROPEAN JOURNAL OF HUMAN GENETICSIF4.62018-05-1024OAAI
Structural determinants and cellular environment define processed actin as the sole substrate of the N-terminal acetyltransferase NAA80PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICAIF9.12018-03-2661OAAI
Expanding the Phenotype Associated with NAA10-Related N-Terminal Acetylation DeficiencyHUMAN MUTATIONIF3.72016-05-0471OA
NAA10 mutation causing a novel intellectual disability syndrome with Long QT due to N- terminal acetyltransferase impairmentSCIENTIFIC REPORTSIF3.92015-11-0265OAAI
An Organellar Nα-Acetyltransferase, Naa60, Acetylates Cytosolic N Termini of Transmembrane Proteins and Maintains Golgi IntegrityCELL REPORTSIF6.92015-03-01121OAAI
Biochemical and cellular analysis of Ogden syndrome reveals downstream Nt-acetylation defectsHUMAN MOLECULAR GENETICSIF3.22014-12-08109OAAI
De novo missense mutations in the NAA10 gene cause severe non-syndromic developmental delay in males and femalesEUROPEAN JOURNAL OF HUMAN GENETICSIF4.62014-08-0668OAAI
A Saccharomyces cerevisiae Model Reveals In Vivo Functional Impairment of the Ogden Syndrome N-Terminal Acetyltransferase NAA10 Ser37Pro MutantMOLECULAR & CELLULAR PROTEOMICSIF5.52014-08-0142OAAI
Protein N-terminal Acetyltransferases Act as N-terminal Propionyltransferases In Vitro and In VivoMOLECULAR & CELLULAR PROTEOMICSIF5.52013-01-0130OAAI