Shigeki Suzuki

dentistry and pharmaceutical sciences

H指数:22

被引数:3,132

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Smad4 and p65 integration at Samd9l regulatory elements shapes BMP protection against inflammatory cell death

Smad4和p65在Samd9l调控元件上的整合塑造了BMP对炎性细胞死亡的保护作用

Communications Biology

IF5.1

2026-09-010

Alpha-Ketoglutarate Drives an Osteogenic and Extracellular Matrix Gene Program in Periodontal Ligament Fibroblasts via Selective Reduction of H3K27me3

α-酮戊二酸通过选择性降低H3K27me3，驱动牙周膜成纤维细胞的成骨和细胞外基质基因程序。

Biology

IF3.5

2026-02-240

OAAI

Establishment of a regenerative endodontic procedures model of mature mouse teeth and evaluation of the wound healing process

建立成熟小鼠牙齿再生性牙髓治疗模型及评估伤口愈合过程

ODONTOLOGY

IF2.4

2025-09-010

OAAI

Piezo1-mediated mechanotransduction in cementocytes via protein kinase B and p38 mitogen-activated protein kinase signaling

通过蛋白激酶B和p38丝裂原活化蛋白激酶信号通路介导的成牙骨质细胞Piezo1机械转导

Journal of Dental Sciences

IF3.1

2025-08-140

Periodontal ligament fibroblasts utilize isoprenoid intermediate farnesyl diphosphate for maintaining osteo/ cementogenic differentiation abilities

JOURNAL OF DENTAL SCIENCES

IF3.1

2025-01-010

OAAI

Periodontal inflammation potentially inhibits hepatic cytochrome P450 expression and disrupts the omega-3 epoxidation pathway in a murine model

JOURNAL OF DENTAL SCIENCES

IF3.1

2025-01-010

OAAI

Cementocyte-derived extracellular vesicles regulate osteoclastogenesis and osteoblastogenesis

牙骨质细胞衍生的细胞外囊泡调节破骨细胞生成和成骨细胞生成

JOURNAL OF DENTAL SCIENCES

IF3.1

2024-10-012

OAAI

A triple growth factor strategy for optimizing bone augmentation in mice

JOURNAL OF BIOMEDICAL MATERIALS RESEARCH PART B-APPLIED BIOMATERIALS

IF3.4

2024-07-120

OAAI

The histone deacetylase inhibitor MS-275 enhances the matrix mineralization of dental pulp stem cells by inducing fibronectin expression

组蛋白去乙酰化酶抑制剂MS-275通过诱导纤维连接蛋白表达增强牙髓干细胞的基质矿化

JOURNAL OF DENTAL SCIENCES

IF3.1

2024-07-014

OAAI

Dynamic changes in chromatin accessibility during the differentiation of dental pulp stem cells reveal that induction of odontogenic gene expression is linked with specific enhancer construction

牙髓干细胞分化过程中染色质可及性的动态变化揭示了牙源性基因表达的诱导与特异性增强子的构建有关

JOURNAL OF DENTAL SCIENCES

IF3.1

2024-07-011

OAAI

Procyanidin B2 enhances anti-inflammatory responses of periodontal ligament cells by inhibiting the dominant negative pro-inflammatory isoforms of peroxisome proliferator-activated receptor g

JOURNAL OF DENTAL SCIENCES

IF3.1

2024-07-013

OAAI

Public RNA-seq data-based identification and functional analyses reveal that MXRA5 retains proliferative and migratory abilities of dental pulp stem cells

SCIENTIFIC REPORTS

IF3.9

2023-09-204

OAAI

Epigenetics in susceptibility, progression, and diagnosis of periodontitis

JAPANESE DENTAL SCIENCE REVIEW

IF6.6

2022-11-0124

OAAI

Loss of IκBζ Drives Dentin Formation via Altered H3K4me3 Status

JOURNAL OF DENTAL RESEARCH

IF5.9

2022-02-2210

PRE

Extracellular Vesicles Derived From Murine Cementoblasts Possess the Potential to Increase Receptor Activator of Nuclear Factor-κB Ligand-Induced Osteoclastogenesis

FRONTIERS IN PHYSIOLOGY

IF3.4

2022-02-144

OAAI

Mouse Model of Loeys-Dietz Syndrome Shows Elevated Susceptibility to Periodontitis via Alterations in Transforming Growth Factor-Beta Signaling

FRONTIERS IN PHYSIOLOGY

IF3.4

2021-08-113

OAAI

Inhibition of the CXCL9-CXCR3 axis suppresses the progression of experimental apical periodontitis by blocking macrophage migration and activation

SCIENTIFIC REPORTS

IF3.9

2021-01-2828

OAAI

DMP-1 promoter-associated antisense strand non-coding RNA, panRNA-DMP-1, physically associates with EGFR to repress EGF-induced squamous cell carcinoma migration

MOLECULAR AND CELLULAR BIOCHEMISTRY

IF3.7

2021-01-0910

PREAI

A small nuclear acidic protein (MTI-II, Zn2+-binding protein, parathymosin) attenuates TNF-α inhibition of BMP-induced osteogenesis by enhancing accessibility of the Smad4-NF-κB p65 complex to Smad binding element

MOLECULAR AND CELLULAR BIOCHEMISTRY

IF3.7

2020-04-1811

PREAI

Cyclic Stretch Force Induces Periodontal Ligament Cells to Secrete Exosomes That Suppress IL-1β Production Through the Inhibition of the NF-κB Signaling Pathway in Macrophages

FRONTIERS IN IMMUNOLOGY

IF5.9

2019-06-2081

OAAI

Shigeki Suzuki

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