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Ettore Dolcetti

University of Genoa
H指数:16
被引数:859

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High-fat diet promotes T cell–mediated synaptic dysfunction in multiple sclerosis

高脂饮食促进多发性硬化中T细胞介导的突触功能障碍

2026-09-230
OAAI
CSF Cytokine Network Organization Predicts Progression Independent of Relapse and MRI Activity in Multiple Sclerosis

脑脊液细胞因子网络组织预测多发性硬化症的进展,独立于复发和MRI活动

2026-09-030
OAAI
Physical exercise modulates T cell activity and mitigates synaptic dysfunction in multiple sclerosis through vagus nerve engagement (vol 135, 106523, 2026)

运动调节T细胞活性并通过迷走神经参与缓解多发性硬化中的突触功能障碍(卷135,106523,2026)

2026-09-010
PRE
Physical exercise modulates T cell activity and mitigates synaptic dysfunction in multiple sclerosis through vagus nerve engagement

体育锻炼通过迷走神经的参与调节T细胞活性并减轻多发性硬化中的突触功能障碍。

2026-03-020
PRE
IL-1β contributes to neurological disability in NMOSD AQP4 + Patients

IL-1β在NMOSD AQP4+患者中导致神经功能障碍。

2025-10-080
OA
Interleukin-10 contrasts inflammatory synaptopathy and central neurodegenerative damage in multiple sclerosis

Interleukin-10对比多发性硬化症中的炎性突触病和中枢神经退行性损伤

2024-08-070
OAAI
Re-emergence of T lymphocyte-mediated synaptopathy in progressive multiple sclerosis
2024-06-070
OAAI
Interleukin-9 protects from microglia- and TNF-mediated synaptotoxicity in experimental multiple sclerosis
2024-05-141
OAAI
Distinct intrathecal inflammatory signatures following relapse and anti-COVID-19 mRNA vaccination in multiple sclerosis

多发性硬化症复发和anti-COVID-19 mRNA疫苗接种后的鞘内炎症特征

2023-09-122
PRE
Osteopontin Is Associated with Multiple Sclerosis Relapses

骨桥蛋白与多发性硬化复发有关

2023-01-118
OAAI
Preventive exercise attenuates IL-2-driven mood disorders in multiple sclerosis
2022-10-0116
OAAI
Fatigue in Multiple Sclerosis Is Associated with Reduced Expression of Interleukin-10 and Worse Prospective Disease Activity
2022-08-2313
OAAI
Exercise protects from hippocampal inflammation and neurodegeneration in experimental autoimmune encephalomyelitis
2021-11-0127
OAAI
MiR-142-3p regulates synaptopathy-driven disease progression in multiple sclerosis

MiR-142-3p调节多发性硬化症中突触驱动的疾病进展

2021-10-0621
OAAI