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Tobias Ruck

Ruhr University Bochum
H指数:45
被引数:7,343

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Clinical heterogeneity in chronic inflammatory demyelinating polyneuropathy: data-driven subtypes challenge current classification

慢性炎性脱髓鞘多发性神经病中的临床异质性:数据驱动的亚型挑战现行分类

2026-10-220
OAAI
Practical Guidance on Initiating and Switching Targeted Immunotherapies in Generalised Myasthenia Gravis: A German–Austrian Expert Opinion Paper

关于在全身性重症肌无力中启动和转换靶向免疫疗法:一项德奥专家意见论文

2026-09-100
OAAI
An integrated reference atlas of human skeletal muscle

人类骨骼肌综合参考图谱

2026-08-310
OAAI
Complement Inhibition Therapy in Myasthenic Crisis—A Multicentre Retrospective Analysis of 17 Cases From Germany

重症肌无力危象中的补体抑制疗法——来自德国的17例多中心回顾性分析

2026-04-150
OA
Serum levels of neurofilament light chain (NfL) and glial fibrillary acid protein (GFAP) in patients with asymptomatic and symptomatic carotid artery stenosis
2026-01-012
OA
Cellular and humoral vaccination response under immunotherapies—German consensus on vaccination strategies in neurological autoimmune diseases

细胞和体液疫苗接种反应在免疫疗法下——德国关于神经自身免疫性疾病疫苗接种策略的共识

2025-12-120
OA
Exercise facilitates post-stroke recovery through mitigation of neuronal hyperexcitability via interleukin-10 signaling
2025-10-080
OAAI
Reduction in oral glucocorticoid use after efgartigimod initiation in clinical practice among patients with generalized myasthenia gravis

在临床实践中,重症肌无力全身型患者在使用efgartigimod起始治疗后口服糖皮质激素使用量减少

2025-08-110
OAAI
Dominant rhabdomyolysis linked to a recurrent ATP2A2 variant reducing SERCA2 function in muscle

显性肌溶解症与复发ATP2A2变异相关,该变异降低了肌肉中SERCA2的功能

BRAIN
IF11.7
2025-05-010
OAAI
Lymphocyte signatures correspond to clinical phenotypes in autoimmune limbic encephalitis

淋巴细胞特征与自身免疫性边缘性脑炎的临床表型相对应

2025-04-180
OA
Mitochondrial damage is associated with an early immune response in inclusion body myositis

线粒体损伤与包涵体肌炎的早期免疫反应相关

Brain
IF11.7
2025-04-070
PRE
Stroke aftercare in Germany: findings from an online survey in the outpatient setting of a neurovascular network

德国卒中后护理:一项在神经血管网络门诊环境中的在线调查结果

2025-03-120
OA
Positive allosteric modulation of AMPA receptors via PF4778574 leads to reduced demyelination and clinical disability in experimental models of multiple sclerosis

PF4778574对AMPA受体的正向变构调节可减轻实验性多发性硬化模型中的脱髓鞘和临床残疾。

2025-03-050
OA