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C

Corey L. Anderson

Department of Medicine

H指数:20

被引数:2,042

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Large-scale functional assessment of variants of the potassium channel Kir2.1: clinical and comparative insights

钾通道Kir2.1变体的大规模功能评估：临床与比较见解

Journal of Biological Chemistry

IF3.9

2025-11-260

OAAI

The role of iPSC research for insight into inherited arrhythmia conditions

iPSC研究对于深入了解遗传性心律失常的作用

IF5.7

2025-05-310

OAAI

Atomic-level investigation of KCNJ2 mutations associated with ventricular arrhythmic syndrome phenotypes

SCIENTIFIC REPORTS

IF3.9

2025-04-020

OAAI

Modeling Idiopathic Ventricular Fibrillation Using iPSC Cardiomyocytes and Computational Approaches A Proof-of-Concept Study

JACC-CLINICAL ELECTROPHYSIOLOGY

IF7.7

2024-12-011

OAAI

Engineered Platform to Uncover Cellular Cross-Talk and Arrhythmogenesis in Idiopathic Ventricular Fibrillation (IVF)

CIRCULATION RESEARCH

IF16.2

2024-08-020

PRE

Functional impact of phosphorylation-site mutations on PKA response of Kir2.1

CIRCULATION RESEARCH

IF16.2

2024-08-020

PRE

Global Proteomic Analysis Reveals Alterations in Differentially Expressed Proteins between Cardiopathic Lamin A/C Mutations

JOURNAL OF PROTEOME RESEARCH

IF3.6

2024-05-081

OA

Long QT Syndrome KCNH2 Variant Induces hERG1a/1b Subunit Imbalance in Patient-Specific Induced Pluripotent Stem Cell-Derived Cardiomyocytes

CIRCULATION-ARRHYTHMIA AND ELECTROPHYSIOLOGY

IF9.8

2021-04-0112

OA

A rapid solubility assay of protein domain misfolding for pathogenicity assessment of rare DNA sequence variants

GENETICS IN MEDICINE

IF6.2

2020-10-019

OAAI

Visualizing Mutation-Specific Differences in the Trafficking-Deficient Phenotype of Kv11.1 Proteins Linked to Long QT Syndrome Type 2

FRONTIERS IN PHYSIOLOGY

IF3.4

2018-05-234

OAAI

Functional Invalidation of Putative Sudden Infant Death Syndrome-Associated Variants in the KCNH2-Encoded Kv11.1 Channel

CIRCULATION-ARRHYTHMIA AND ELECTROPHYSIOLOGY

IF9.8

2018-05-014

OAAI

Trafficking Deficient KV11.1 (HERG) Mutations Linked to Long QT Syndrome Localize to Different Endoplasmic Reticulum Subcompartments

BIOPHYSICAL JOURNAL

IF3.1

2016-02-010

OAAI

Mouse ERG K+ Channel Clones Reveal Differences in Protein Trafficking and Function

JOURNAL OF THE AMERICAN HEART ASSOCIATION

IF5.3

2014-12-171

OAAI

Large-scale mutational analysis of Kv11.1 reveals molecular insights into type 2 long QT syndrome

NATURE COMMUNICATIONS

IF15.7

2014-11-24149

OAAI

Pharmacological correction of long QT-linked mutations in KCNH2 (hERG) increases the trafficking of Kv11.1 channels stored in the transitional endoplasmic reticulum

AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY

IF4.7

2013-11-0127

OAAI

Drug-induced long QT syndrome:: hERG K+ channel block and disruption of protein trafficking by fluoxetine and norfluoxetine

药物引起的长QT综合征:: 氟西汀和去甲氟西汀对hERG K通道的阻滞和蛋白质运输的破坏

BRITISH JOURNAL OF PHARMACOLOGY

IF7.7

2009-01-29187

OAAI

Most LQT2 mutations reduce Kv11.1 (hERG) current by a class 2 (trafficking-deficient) mechanism

IF38.6

2006-01-24387

OA

Intragenic suppression of trafficking-defective KCNH2 channels associated with long QT syndrome

MOLECULAR PHARMACOLOGY

IF3

2005-07-0125

PREAI

Thapsigargin selectively rescues the trafficking defective LQT2 channels G601S and F805C

JOURNAL OF BIOLOGICAL CHEMISTRY

IF3.9

2003-09-0197

OAAI

E4031 rescue of HERG does not require the restoration of N-linked glycosyoltion

JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY

IF4.7

2002-07-010

PREAI