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熊

熊伟 (Wei Xiong)

University of Science and Technology of China
H指数:29
被引数:3,203

个人简介

"Our lab has been focusing in the field of neurochemical mechanism of brain functions and brain diseases for a long period. We are developing the multi-scale technology platforms for research of brain small molecules such as single-cell mass spectrometry, single-organelle mass spectrometry, mass spectrometry imaging. Our studies focus on the changes of small molecule metabolites in the brain during aging and neurodegeneration, and the roles of these changed small molecules in the brain aging and neurodegeneration. We are investigating the neuronal circuit mechanism of sensory (especially pain) processing and its related neurological disorders, such as chronic inflammatory pain and allodynia and hyperalgesia in neuropathic pain. We are also developing new therapeutic medicine and treatment to relieve chronic pain. EDUCATION AND RESEARCH EXPERIENCE 03/2013- present Professor, University of Science and Technology of China 10/2011-03/2013 Research Fellow, National Institute on Alcohol Abuse and Alcoholism, National Institutes of Health, Bethesda, Maryland, USA 10/2006-10/2011 Postdoctoral Fellow, National Institute on Alcohol Abuse and Alcoholism, National Institutes of Health, Bethesda, Maryland, USA 09/2001-07/2006 Ph.D., School of Life Sciences, Peking University 09/1997-07/2001 B.S., School of Life Sciences, Peking University"

教育背景

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工作经历

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荣誉奖项

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The activation of catecholamine neurons in the rostral ventrolateral medulla drives ventricular remodeling after myocardial ischemia/reperfusion injury

延髓头端腹外侧区儿茶酚胺能神经元的激活驱动心肌缺血/再灌注损伤后的心室重构

2025-07-080
PRE
Short-term high-dose gemcitabine induces PERK-mediated immunogenic cell death and potentiates antitumor immunity in bladder cancer

短期高剂量吉西他滨诱导PERK介导的免疫原性细胞死亡,并增强膀胱癌的抗肿瘤免疫。

2025-06-200
OA
BACE1-dependent cleavage of GABAA receptor contributes to neural hyperexcitability and disease progression in Alzheimer's disease

BACE1依赖性的GABAA受体裂解有助于神经兴奋性过高和阿尔茨海默病病情进展

NEURON
IF15
2025-02-013
PRE
PAFAH2 suppresses synchronized ferroptosis to ameliorate acute kidney injury (FEB, 10.1038/s41589-023-01528-7, 2023)
2024-02-050
OA
PAFAH2 suppresses synchronized ferroptosis to ameliorate acute kidney injury
2024-01-2911
PRE