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Fortunata Carbone

Institute for Experimental Endocrinology and Oncology
H指数:26
被引数:3,072

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High-fat diet promotes T cell–mediated synaptic dysfunction in multiple sclerosis

高脂饮食促进多发性硬化中T细胞介导的突触功能障碍

2026-09-230
OAAI
Physical exercise modulates T cell activity and mitigates synaptic dysfunction in multiple sclerosis through vagus nerve engagement (vol 135, 106523, 2026)

运动调节T细胞活性并通过迷走神经参与缓解多发性硬化中的突触功能障碍(卷135,106523,2026)

2026-09-010
PRE
CCL2: A Pro-Inflammatory Driver and Candidate Diagnostic Biomarker in Colorectal Cancer Patients

CCL2:结直肠癌患者中的一种促炎驱动因子和候选诊断生物标志物

2026-07-230
OAAI
MicroRNA-142-3p shuttling in extracellular vesicles marks regulatory T cell dysfunction in multiple sclerosis

MicroRNA-142-3p在细胞外囊泡中的穿梭标志着多发性硬化中调节性T细胞的功能障碍

2025-01-010
PRE
Deciphering the role of protein kinase A in the control of FoxP3 expression in regulatory T cells in health and autoimmunity
2024-07-300
OAAI
Osteopontin Is Associated with Multiple Sclerosis Relapses

骨桥蛋白与多发性硬化复发有关

2023-01-118
OAAI
High levels of blood circulating immune checkpoint molecules in children with new-onset type 1 diabetes are associated with the risk of developing an additional autoimmune disease

新发1型糖尿病患儿血液循环免疫检查点分子水平高与发生其他自身免疫性疾病的风险相关

2022-05-253
OA
A novel smaller β-defensin-derived peptide is active against multidrug-resistant bacterial strains

一种新型的较小的 β-防御素衍生肽对多重耐药细菌菌株具有活性

2021-11-246
OA
Signals of pseudo-starvation unveil the amino acid transporter SLC7A11 as key determinant in the control of Treg cell proliferative potential

伪饥饿信号揭示了氨基酸转运蛋白SLC7A11作为控制Treg细胞增殖潜能的关键决定因素

IMMUNITY
IF26.3
2021-07-0149
OA
Randomised Clinical Trial: Calorie Restriction Regimen with Tomato Juice Supplementation Ameliorates Oxidative Stress and Preserves a Proper Immune Surveillance Modulating Mitochondrial Bioenergetics of T-Lymphocytes in Obese Children Affected by Non-Alcoholic Fatty Liver Disease (NAFLD)
2020-01-0427
OAAI
Obesity worsens central inflammation and disability in multiple sclerosis
2019-06-0482
PREAI
Immunometabolic profiling of T cells from patients with relapsing-remitting multiple sclerosis reveals an impairment in glycolysis and mitochondrial respiration
2017-12-0170
OAAI