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Unanticipated specificity in effector-triggered immunity
DOI:10.1016/j.it.2024.10.008.png)
摘要
En 中文
Effector-triggered immunity (ETI) enables hosts to react to pathogens by monitoring few key cellular processes. ETI responses are assumed to be similar toward related pathogen effectors. However, recent evidence from the invertebrate model Caenorhabditis elegans and pore- forming toxins indicates a much more complex and specific ETI than previously anticipated.
Keyword:
PATHWAYS
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期刊
IF:
13.9
论文数:
2.7K
被引数:
1.7W
机构
引用论文
Aminopeptidase MNP-1 triggers intestine protease production by activating daf-16 nuclear location to degrade pore-forming toxins in Caenorhabditis elegans
PLOS PATHOGENS
IF4.9
Bacillus thuringiensis Crystal Protein Cry6Aa Triggers Caenorhabditis elegans Necrosis Pathway Mediated by Aspartic Protease (ASP-1)
PLOS PATHOGENS
IF4.9

