H
Hyungjin Eoh
H-index:27
Citation Count:2,769
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KSHV vIRF3–PKM2 interaction induces SMAD5 acetylation, promoting Kaposi’s sarcoma progression
KSHV vIRF3–PKM2相互作用诱导SMAD5乙酰化,促进卡波西肉瘤进展
Retraction Note: NOTCH localizes to mitochondria through the TBC1D15-FIS1 interaction and is stabilized via blockade of E3 ligase and CDK8 recruitment to reprogram tumor-initiating cells
撤稿声明:NOTCH通过TBC1D15-FIS1相互作用定位于线粒体,并通过阻断E3连接酶和CDK8招募来重编程肿瘤起始细胞
Transient drug-tolerance and permanent drug-resistance rely on the trehalose-catalytic shift in Mycobacterium tuberculosis
短暂的药物耐受性和永久性耐药性依赖于结核分枝杆菌的海藻糖催化转变
NATURE COMMUNICATIONS
IF15.7

