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Molecular Medicine 封面图

Molecular Medicine

IF6.4
论文数3286
被引数8311
期刊论文 3286
发表时间
TGF-β, but not IL-4, polarized macrophages induce fibrotic responses in lung fibroblastsTGF-β,而非IL-4,极化的巨噬细胞诱导肺成纤维细胞的纤维化反应
delete2026-10-30
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deleteQian Tian; Alexandra Drakaki; Hella L. Aberson; Sami Lone; Tom van der Poll; Marten A. Hoeksema; C. Arnold Spek; Jan Willem Duitman
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Adipose Cirbp protects against diet-induced obesity by regulating Vav3 to promote brown fat thermogenesis脂肪组织Cirbp通过调控Vav3促进棕色脂肪产热,以抵抗饮食诱导的肥胖
delete2026-10-13
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deleteShaojun Huang; Shiyi Tan; Xiaowan Jiang; Yuanyuan Liu; Zijian Chen; Xudong Mai; Jitong Li; Weiheng Wen; Hong Chen
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Upregulation of FAT10 by TLR4/NF-kappaB pathway contributes to MASLD development by the modulation of FASN stability and mitophagy通过调节FASN稳定性和线粒体自噬,TLR4/NF-κB通路上调FAT10表达促进MASLD的发生发展
delete2026-10-12
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deleteWeiliang Zhang; Jianwei Hong; Weirong Lu; Tianhao Li; Haokun Di; Chunyue Wang; Junyang Pan; Lishuai Qu; Jianya Zhao; Ruoyu Zhou; Jinxia Liu; Chunhua Wan
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CD24 blockade reprograms macrophages and synergizes with CD47 blockade to overcome tumor immune evasionCD24阻断重编程巨噬细胞并与CD47阻断协同作用以克服肿瘤免疫逃逸
delete2026-10-09
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deleteGuocai Tian; You Fu; Hui Zeng; Linjuan Du; Lujun Zhou; Xuefeng Pan; Ting Dong; Shuo Xu; Boyan Chen; Xuebing Bai; Xiaoqian Meng; Yulin Li; Wenzhen Lin; Zhiyuan Zhang; Shengzhong Duan; Xiaogang Pan; Yan-an Wang; Wuchang Zhang
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Pharmacological GPR39 activation protects against MASLD/MASH and improves metabolic syndrome features: a preclinical study in diet-induced mouse models药理学GPR39激活可预防MASLD/MASH并改善代谢综合征特征:一项基于饮食诱导小鼠模型的临床前研究
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deleteAnna Laitakari; Jens C. B. Jacobsen; Zilu Ye; Steen S. Poulsen; Joyceline Cuenco; Sabrina Klotz; Anna S. Hassing; Lingzhi Liu; Chunyu Lin; Teeradon Phlairaharn; Costanza M. Cucchiara; Annemette O. Brethvad; Anders Bach; Birgitte Holst; Jonas T. Treebak; Jesper V. Olsen; Kei Sakamoto; Thomas M. Frimurer
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Post-translational modifications in sepsis: mechanisms and therapeutic potential in macrophage polarization脓毒症中的翻译后修饰:巨噬细胞极化机制及治疗潜力
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deleteHuang Wu; Yan Liao; Lingxuan Tang; Lizhou Song; Changli Wang; Haoling Zhang; Xiaoming Deng; Wangzheqi Zhang; Lulong Bo
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