未登录 Loss of Socs2 improves molecular responses to IFNα in a mouse model of myeloproliferative neoplasms driven by JAK2-V617F 在由JAK2-V617F驱动的骨髓增殖性肿瘤小鼠模型中,Socs2的缺失改善了IFNα的分子应答。 Usart, Marc; Kimmerlin, Quentin; Stetka, Jan; Stoll, Cedric; Rai, Shivam; Fonseca, Tiago Almeida; Karjalainen, Riikka; Hao-Shen, Hui; Roux, Julien; El Taher, Athimed; Lynch, Dylan; Makukhin, Nikolai; Ciulli, Alessio; Skoda, Radek C. 分享 收藏
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Design of a Cereblon construct for crystallographic and biophysical studies of protein degraders Kroupova, Alena; Spiteri, Valentina A.; Rutter, Zoe J.; Furihata, Hirotake; Darren, Darren; Ramachandran, Sarath; Chakraborti, Sohini; Haubrich, Kevin; Pethe, Julie; Gonzales, Denzel; Wijaya, Andre J.; Rodriguez-Rios, Maria; Sturbaut, Manon; Lynch, Dylan M.; Farnaby, William; Nakasone, Mark A.; Zollman, David; Ciulli, Alessio 分享 收藏
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Structure-based design of a phosphotyrosine-masked covalent ligand targeting the E3 ligase SOCS2 Ramachandran, Sarath; Makukhin, Nikolai; Haubrich, Kevin; Nagala, Manjula; Forrester, Beth; Lynch, Dylan M.; Casement, Ryan; Testa, Andrea; Bruno, Elvira; Gitto, Rosaria; Ciulli, Alessio 分享 收藏
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