未登录

Z

Zhuoya Yu

Inner Mongolia Agricultural University

H指数:7

被引数:334

个人简介

暂无内容

教育背景

暂无内容

工作经历

暂无内容

荣誉奖项

暂无内容

An underlying mechanism of bovine mastitis: PGE2 regulates Staphylococcus aureus-induced inflammatory response through TLR2, TLR4, and NLRP3 in macrophages

牛乳腺炎的潜在机制：PGE2通过TLR2、TLR4和NLRP3调节巨噬细胞中金黄色葡萄球菌诱导的炎症反应

Veterinary Quarterly

IF5.2

2026-05-231

PRE

Molecular mechanism of drug inhibition of URAT1

药物抑制URAT1的分子机制

Nature Communications

IF15.7

2025-07-160

OA

MLKL is involved in the regulation of skin wound healing and interplay between macrophages and myofibroblasts in mice

MLKL参与小鼠皮肤伤口愈合的调控以及巨噬细胞与肌成纤维细胞之间的相互作用。

SCIENTIFIC REPORTS

IF3.9

2025-04-190

OA

Molecular insights into the α6β4 nicotinic acetylcholine receptor function and ligand recognition

α6β4烟碱乙酰胆碱受体功能和配体识别的分子见解

NATURE COMMUNICATIONS

IF15.7

2025-04-020

OA

Matrine alleviates Staphylococcus aureus-induced acute lung injury in mice by inhibiting MLKL and NLRP3-mediated inflammatory activity

EUROPEAN JOURNAL OF PHARMACOLOGY

IF4.7

2025-04-010

PRE

Taurochenodeoxycholic acid ameliorates the Staphylococcus aureus infection-induced acute lung injury through toll-like receptor 2 in mice

INTERNATIONAL IMMUNOPHARMACOLOGY

IF4.7

2024-12-010

PRE

Structural snapshots of human VMAT2 reveal insights into substrate recognition and proton coupling mechanism

IF25.9

2024-05-220

PRE

Structural bases of inhibitory mechanism of CaV1.2 channel inhibitors

NATURE COMMUNICATIONS

IF15.7

2024-03-303

OAAI

Prostaglandin D2 is involved in the regulation of inflammatory response in Staphylococcus aureus-infected mice macrophages

INTERNATIONAL IMMUNOPHARMACOLOGY

IF4.7

2024-03-010

PRE

Structural insight into the allosteric inhibition of human sodium-calcium exchanger NCX1 by XIP and SEA0400

XIP和SEA0400对人类钠钙交换体NCX1的变构抑制的结构洞察

IF8.3

2023-12-155

OAAI

Structural basis of ligand binding modes of human EAAT2

NATURE COMMUNICATIONS

IF15.7

2022-06-0915

OAAI

Structure, gating, and pharmacology of human Cav3.3 channel

NATURE COMMUNICATIONS

IF15.7

2022-04-1926

OAAI

Closed-state inactivation and pore-blocker modulation mechanisms of human CaV2.2

IF6.9

2021-11-0143

OA