Anna Shteinfer-Kuzmine

Ben-Gurion University of the Negev

H指数:19

被引数:2,335

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MAVS as a Key Regulator of Tumor Proliferation, Survival, the Tumor Microenvironment, and Immunity

MAVS作为肿瘤增殖、生存、肿瘤微环境和免疫的关键调控因子

Biomolecules

IF4.8

2026-03-300

OAAI

VDAC1-interacting proteins: binding site mapping and their derived peptides induce apoptosis and multifaceted cellular effects

VDAC1相互作用蛋白：结合位点映射及其衍生的肽诱导凋亡和多种细胞效应

Apoptosis

IF8.1

2025-09-260

OAAI

Metformin-Induced Apoptosis Is Mediated Through Mitochondrial VDAC1

二甲双胍诱导的凋亡通过线粒体VDAC1介导

PHARMACEUTICALS

IF4.8

2025-05-200

Elevated serum mtDNA in COVID-19 patients is linked to SARS-CoV-2 envelope protein targeting mitochondrial VDAC1, inducing apoptosis and mtDNA release (vol 29, pg 2025, 2024)

COVID-19患者血清中升高的线粒体mtDNA与SARS-CoV-2包膜蛋白靶向线粒体VDAC1、诱导凋亡及mtDNA释放相关（卷29，页2025，2024）。

APOPTOSIS

IF8.1

2025-05-100

Resveratrol's Pro-Apoptotic Effects in Cancer Are Mediated Through the Interaction and Oligomerization of the Mitochondrial VDAC1

白藜芦醇在癌症中的促凋亡效应是通过线粒体VDAC1的相互作用和寡聚化介导的。

INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES

IF4.9

2025-04-220

Elevated serum mtDNA in COVID-19 patients is linked to SARS-CoV-2 envelope protein targeting mitochondrial VDAC1, inducing apoptosis and mtDNA release

新型冠状病毒肺炎患者血清mtDNA升高与靶向线粒体VDAC1的SARS-CoV-2包膜蛋白，诱导细胞凋亡和mtDNA释放有关

APOPTOSIS

IF8.1

2024-10-071

OAAI

Interaction of SMAC with a survivin-derived peptide alters essential cancer hallmarks: Tumor growth, inflammation, and immunosuppression

MOLECULAR THERAPY

IF12

2024-06-012

OAAI

Silencing the Mitochondrial Gatekeeper VDAC1 as a Potential Treatment for Bladder Cancer

CELLS

IF5.2

2024-04-043

OAAI

The role of the mitochondrial protein VDAC1 in inflammatory bowel disease: a potential therapeutic target (vol 30, pg 726, 2022)

MOLECULAR THERAPY

IF12

2024-04-010

Overexpression of the mitochondrial anti-viral signaling protein, MAVS, in cancers is associated with cell survival and inflammation

MOLECULAR THERAPY NUCLEIC ACIDS

IF6.1

2023-09-016

OAAI

Apoptotic proteins with non-apoptotic activity: expression and function in cancer

APOPTOSIS

IF8.1

2023-04-0428

OAAI

Targeting the overexpressed mitochondrial protein VDAC1 in a mouse model of Alzheimer's disease protects against mitochondrial dysfunction and mitigates brain pathology (vol 11, 58, 2022)

TRANSLATIONAL NEURODEGENERATION

IF15.2

2023-01-110

OAAI

Targeting the overexpressed mitochondrial protein VDAC1 in a mouse model of Alzheimer's disease protects against mitochondrial dysfunction and mitigates brain pathology

TRANSLATIONAL NEURODEGENERATION

IF15.2

2022-12-2838

OAAI

Non-apoptotic activity of the mitochondrial protein SMAC/Diablo in lung cancer: Novel target to disrupt survival, inflammation, and immunosuppression

FRONTIERS IN ONCOLOGY

IF3.3

2022-09-147

OAAI

The role of the mitochondrial protein VDAC1 in inflammatory bowel disease: a potential therapeutic target

MOLECULAR THERAPY

IF12

2022-02-0153

OAAI

Adverse Effects of Metformin From Diabetes to COVID-19, Cancer, Neurodegenerative Diseases, and Aging: Is VDAC1 a Common Target?

FRONTIERS IN PHYSIOLOGY

IF3.4

2021-10-0427

OAAI

VDAC1 Silencing in Cancer Cells Leads to Metabolic Reprogramming That Modulates Tumor Microenvironment

癌细胞中的VDAC1沉默导致调节肿瘤微环境的代谢重编程

CANCERS

IF4.4

2021-06-0715

OAAI

SMAC/Diablo controls proliferation of cancer cells by regulating phosphatidylethanolamine synthesis

SMAC/Diablo通过调节磷脂酰乙醇胺的合成来控制癌细胞的增殖

MOLECULAR ONCOLOGY

IF4.5

2021-05-0413

OAAI

Mitochondria and nucleus cross-talk: Signaling in metabolism, apoptosis, and differentiation, and function in cancer

IUBMB LIFE

IF3.2

2020-11-1223

OAAI

A VDAC1-Derived N-Terminal Peptide Inhibits Mutant SOD1-VDAC1 Interactions and Toxicity in the SOD1 Model of ALS

FRONTIERS IN CELLULAR NEUROSCIENCE

IF4

2019-08-1428

OAAI

Anna Shteinfer-Kuzmine

个人简介

教育背景

工作经历

荣誉奖项

研究方向